Complications of pressure sores
Complications can occur from ongoing
pressure sores and also from treating pressure
sores.1 Some complications of ongoing pressure sores
include:
- Infections.
- Cellulitis is an infection of the skin. Cellulitis
causes painful, red, hot, swollen skin that may crack, split, or weep
fluid.
- Osteomyelitis is an infection in the
bone. It causes swollen and red skin over the affected bone. It also causes
pain that is worse when pressing on the infected area.
- Bacteremia or a
bacterial infection in the blood (sepsis).
-
Necrotizing fasciitis
or a bacterial infection that destroys skin and soft tissues
such as fat and muscle.
-
Endocarditis
,
which is an infection of the heart lining. Symptoms of endocarditis include
vague, flu-like symptoms, such as chronic low-grade fever and fatigue.
-
Meningitis
, which is an infection of the fluid that
surrounds the brain and spine. Meningitis causes a severe headache with a stiff
neck, fever, nausea, and vomiting.
-
Septic arthritis
, which is pain and inflammation of a joint caused by a
bacterial infection. Septic arthritis causes a joint to be red, hot, swollen,
and tender.
-
Abscesses
. An abscess can form on the
skin or on tissues within the body and cause pain, swelling, and
tenderness.
- Deposits of bone in soft tissues (heterotopic bone
formation). These occur especially around the hip joint and can cause pain and
problems with walking or moving the legs.
-
Squamous cell
skin cancer, especially if the wound becomes a
stage 4 pressure sore. Symptoms of squamous cell cancer include growths or
patches of skin that feel scaly, bleed, or develop a crust, and sores that do
not heal.
Citations
-
O'Connor K (2005). Pressure ulcers. In JA DeLisa et
al., eds., Physical Medicine and Rehabilitation: Principles and Practice, 4th ed., vol. 2, pp. 1605–1618. Philadelphia: Lippincott
Williams and Wilkins.
Last Updated:
March 5, 2009
O'Connor K (2005). Pressure ulcers. In JA DeLisa et
al., eds., Physical Medicine and Rehabilitation: Principles and Practice, 4th ed., vol. 2, pp. 1605–1618. Philadelphia: Lippincott
Williams and Wilkins.